Role Of Endothelium-Derived Nitric Oxide In The Modulation Of Canine Myocardial Mitochondrial Respiration In Vitro
The mechanism responsible for the regulation of cardiac function by endogenous nitric oxide (NO) remains unclear. In this investigation, O
consumption by freshly isolated myocardial muscle segments from the left ventricular free wall of canine hearts was quantified by a Clark-type O
electrode at 37°C.